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- W2057607194 abstract "Glucocorticoids bind to hippocampal mineralo-(MR) and gluco-(GR) corticoid receptors and, at high concentrations (e.g. as seen following treatment with pharmacological doses of corticosteroids or during stress), may affect hippocampal neuronal function. Such actions could involve brain-derived neurotrophic factor (BDNF), its receptor, trkB, and the excitatory neurotransmitter, glutamate. This experiment investigated the effect of a single intravenous (i.v.) injection of the synthetic glucocorticoid, dexamethasone (Dex, 5 mg kg–1) on gene expression for MR s,GR s, BDNF, trkB, and selected ionotropic glutamate receptor subunits (iGluRs), in the porcine hippocampus. Quantification of m RNA s in the brains of pigs (n = 4/treatment) killed 24 hours after saline or Dex administration indicated a significant Dex-induced decrease in BDNF m RNA in all hippocampal regions. However, gene expression for MR s, GR s, trkB and iGluRs was unaffected at this time-point." @default.
- W2057607194 created "2016-06-24" @default.
- W2057607194 creator A5069840343 @default.
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- W2057607194 date "2001-04-01" @default.
- W2057607194 modified "2023-10-17" @default.
- W2057607194 title "Down-regulation of BDNF mRNA, with no effect on trkB or glucocorticoid receptor mRNAS, in the porcine hippocampus after acute dexamethasone treatment" @default.
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- W2057607194 doi "https://doi.org/10.1053/rvsc.2001.0456" @default.
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