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• W2057783048 abstract "ABSTRACT Enteropathogenic Escherichia coli (EPEC) and Shiga toxin-producing E. coli (STEC) induce cytoskeletal changes in infected epithelial cells. To further characterize host cytosolic responses to infection, a series of specific cell-signaling inhibitors were employed. Initial bacterial adhesion to HEp-2 epithelial cells was not reduced, whereas α-actinin accumulation in infected cells was blocked by a phosphoinositide-specific phospholipase C inhibitor (ET-18-OCH 3 ), phosphoinositide 3-kinase inhibitors (wortmannin and LY294002), and a 5-lipoxygenase inhibitor, nordihydroguaretic acid. A cyclooxygenase-2 inhibitor (NS-398), however, did not block α-actinin reorganization in response to EPEC and STEC infections. Understanding signal transduction responses to enteric pathogens could provide the basis for the development of novel therapeutic strategies." @default.
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• W2057783048 date "2001-11-01" @default.
• W2057783048 modified "2023-10-02" @default.
• W2057783048 title "Inhibition of Attaching and Effacing Lesion Formation following Enteropathogenic <i>Escherichia coli</i> and Shiga Toxin-Producing <i>E. coli</i> Infection" @default.
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• W2057783048 doi "https://doi.org/10.1128/iai.69.11.7152-7158.2001" @default.
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