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- W2058169110 abstract "To summarize the recent knowledge on the implications of placenta and cytokines in normal and preeclamptic pregnancies.A literature search was conducted of applicable articles related to interactions between trophoblast and cytokines in generating preeclampsia.The initiating event in preeclampsia has been postulated to be the reduced uteroplacental perfusion as a result of abnormal extravillous cytotrophoblast invasion and remodeling of the uterine spiral arteries. Focal ischemia and hypoxia, deportation of hypoxemic trophoblast cells and abnormal expression of various placental biologic molecules, particularly the cytokines, are thought to lead to widespread dysfunction of the maternal vascular endothelium resulting in overproduction of endothelin and thromboxane, enhanced vascular sensitivity to angiotensin II, and reduced secretion of vasodilators such as nitric oxide and prostacyclin. These alterations, in turn, cause hypertension, proteinuria and edema, and pathologies in many organ systems (kidney, lung, liver, brain)." @default.
- W2058169110 created "2016-06-24" @default.
- W2058169110 creator A5007178672 @default.
- W2058169110 creator A5040657551 @default.
- W2058169110 creator A5058217748 @default.
- W2058169110 creator A5077019788 @default.
- W2058169110 creator A5079655790 @default.
- W2058169110 creator A5085143690 @default.
- W2058169110 date "2003-07-01" @default.
- W2058169110 modified "2023-09-30" @default.
- W2058169110 title "Trophoblastic remodeling in normal and preeclamptic pregnancies: implication of cytokines" @default.
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