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- W2058193201 abstract "Prolongation of the QT interval in the electrocardiogram (ECG) occurs in several states in which the incidence of cardiac arrhythmias and sudden death is high. These include the idiopathic prolonged QT interval syndromes, quinidine and various other drug intoxications, and hypokalemia. Such associations demonstrate that QT interval prolongation sometimes identifies cardiac conditions susceptible to serious arrhythmias but detailed mechanisms responsible for the associations have not been determined. It has been shown that the relation of QT interval and ventricular recovery time is complex in that some agencies which alter recovery time result in paradoxical effects on the interval. '-+ One of these agencies is left stellate ganglion stimulation in the dog, which frequently results in prolongation of the QT interval, although refractory periods are actually reduced in the innervated area. 1, 4 This suggests that prolongation of the QT interval may sometimes reflect a cardiac state with greater than normal disparity of recovery times rather than one in which simply prolonged recovery is present. The degree of inequality of recovery times in cardiac muscle has been extensively documented to be a factor in arrhythmia susceptibility2 -'2 These considerations suggest that the utility of the QT interval as an indicator of cardiac states vulnerable to arrhythmias should be further investigated. The present study was undertaken to elucidate mechanisms operating in the reflection of ventricular recovery by the QT interval. Findings" @default.
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- W2058193201 date "1976-08-01" @default.
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- W2058193201 title "Adrenergic effects on the QT interval of the electrocardiogram" @default.
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- W2058193201 doi "https://doi.org/10.1016/s0002-8703(76)80256-6" @default.
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