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- W2058205022 endingPage "126" @default.
- W2058205022 startingPage "117" @default.
- W2058205022 abstract "A turning point of research in Alzheimer’s disease was undoubtedly the discovery of BACE1, the amyloid-β precursor protein-cleaving enzyme that initiates the generation of amyloid-β, the peptide strongly suspected to be responsible for neuronal malfunction and death. Several research groups started a race to identify the best inhibitor of BACE1 activity. On the other hand, basic researchers are evaluating the changes in BACE1 expression and activity with the aim to better understand the pathogenetic process of the disease. Along this second line of research, in the last few years many important results have been reported in various experimental models, as well as in Alzheimer’s disease patients. As a consequence, new pathogenetic paradigms have been developed. We have reviewed these reports trying to highlight contrasting viewpoints, data awaiting final confirmation, and promising perspectives." @default.
- W2058205022 created "2016-06-24" @default.
- W2058205022 creator A5000207843 @default.
- W2058205022 creator A5010982287 @default.
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- W2058205022 creator A5046665241 @default.
- W2058205022 creator A5048276806 @default.
- W2058205022 creator A5054378613 @default.
- W2058205022 creator A5057573761 @default.
- W2058205022 date "2007-01-01" @default.
- W2058205022 modified "2023-09-24" @default.
- W2058205022 title "BACE1 Expression and Activity: Relevance in Alzheimer’s Disease" @default.
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