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- W2058586283 abstract "The mechanisms by which dysfunctional glial cells contribute to ALS have remained poorly defined. Now, Harvard University researchers have identified a therapeutic target—prostaglandin D2 receptor subtype DP1—and shown that reducing its expression can prolong survival in a mouse model of ALS." @default.
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- W2058586283 date "2014-08-01" @default.
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- W2058586283 title "A cultured approach to ALS therapy" @default.
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- W2058586283 doi "https://doi.org/10.1038/scibx.2014.974" @default.
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