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- W2058772405 endingPage "17" @default.
- W2058772405 startingPage "6" @default.
- W2058772405 abstract "Abstract: Pulmonary arterial hypertension (PAH) is a devastating disorder characterized by abnormal increased vasoconstriction and vascular remodelling. In this review we discuss the pathophysiology, genetic basis and clinical features of this disorder. Current therapy of PAH is based on an understanding of its pathogenesis, and we review current treatment options based on the pathophysiology of the disease. We discuss three promising novel therapies studied in animal models and human tissue. All three therapies appear to prevent and reduce pulmonary arterial medial hyperplasia through their anti‐proliferative and/or pro‐apoptotic effects: serotonin transporter inhibitors by blocking serotonin uptake; dichloroacetate by activating voltage‐gated potassium channels; and simvastatin by preventing activation of small GTPases." @default.
- W2058772405 created "2016-06-24" @default.
- W2058772405 creator A5052093274 @default.
- W2058772405 creator A5062800037 @default.
- W2058772405 creator A5071223454 @default.
- W2058772405 date "2006-01-01" @default.
- W2058772405 modified "2023-10-15" @default.
- W2058772405 title "Pulmonary arterial hypertension: New insights and new hope" @default.
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- W2058772405 doi "https://doi.org/10.1111/j.1440-1843.2006.00778.x" @default.
- W2058772405 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/16423196" @default.
- W2058772405 hasPublicationYear "2006" @default.