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• W2058952682 abstract "It is well known that acute cocaine administration increases c-Fos expression that is involved in cocaine-induced persistent changes in the central nervous system. In the present study, we investigated a possible involvement of extracellular signal-regulated protein kinase (ERK) in induction of c-fos expression in response to acute cocaine treatment in nucleus accumbens (NAc). We found that inhibition of ERK activation significantly attenuated cocaine-induced c-fos expression at both protein and mRNA levels in the NAc. Furthermore, using an immunofluorescent staining approach, we found that inhibition of ERK activation completely abolished cocaine-induced increase in number of c-Fos-positive cells in the core region of NAc, whereas, in shell region of NAc, inhibition of ERK activation partially attenuated cocaine-induced c-Fos expression. Our findings suggest that ERK might participate in cocaine-induced c-fos expression in the NAc, particularly in the core region of NAc." @default.
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• W2058952682 date "2008-06-01" @default.
• W2058952682 modified "2023-09-24" @default.
• W2058952682 title "Involvement of extracellular signal-regulated protein kinase in acute cocaine-induced c-fos in nucleus accumbens" @default.
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• W2058952682 doi "https://doi.org/10.1016/j.neulet.2008.04.035" @default.
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