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- W2059095384 abstract "Chronic kidney disease is associated with increased levels of assymetric N(G),N(G)-dimethylarginine (ADMA), which is predictive of increased mortality and cardiovascular disease. ADMA induces endothelial dysfunction through competitive inhibition of the endothelial nitric oxide (eNOS) substrate L-arginine. Kajimoto et al. show that ADMA may also reduce nitric oxide production via decreased eNOS phosphorylation; this effect is mediated by the MAPK pathway and can be reversed in vivo by increased catabolism of ADMA through dimethylarginine dimethylaminohydrolase-1 overexpression." @default.
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- W2059095384 date "2012-04-01" @default.
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- W2059095384 title "ADMA and NOS regulation in chronic renal disease: beyond the old rivalry for l-arginine" @default.
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- W2059095384 doi "https://doi.org/10.1038/ki.2011.496" @default.
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