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• W2059368542 abstract "Based on the associations reviewed in this report, we have hypothesized that retardation of renal development as occurs in individuals of low birth weight gives rise to increased postnatal risks for systemic and glomerular hypertension as well as enhanced risk of expression of renal disease. This hypothesis draws on observations suggesting (1) a direct relationship between birth weight and nephron number, (2) an inverse relationship between birth weight and later-life hypertension, and (3) an inverse relationship between nephron number and blood pressure, irrespective of whether nephron number is reduced congenitally or in postnatal life (as from partial renal ablation or acquired renal disease). Additional clinical and epidemiologic studies are needed to assess these initial impressions." @default.
• W2059368542 created "2016-06-24" @default.
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• W2059368542 date "1994-02-01" @default.
• W2059368542 modified "2023-10-18" @default.
• W2059368542 title "Congenital Oligonephropathy and the Etiology of Adult Hypertension and Progressive Renal Injury" @default.
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• W2059368542 doi "https://doi.org/10.1016/s0272-6386(12)80967-x" @default.
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