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- W2059700757 endingPage "R35" @default.
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- W2059700757 abstract "The signaling pathways activated by the steroid hormone oestrogen include a variety of cytoplasmic second messengers linked to a multitude of tissue-specific effects. In the last decade, sphingolipids and their membrane receptors were added to the list of oestrogen-activated mediators. Oestrogen triggers the sphingolipid signalling cascade in various tissues including breast cancer. Extensive research has shown that sphingolipids are the key regulatory molecules in growth factor networks. Sphingolipids can control the rate of cell proliferation and the differentiation outcome during malignant transformation. In this study, we summarise novel experimental evidences linking sphingolipids to oestrogen-activated effects, highlight the role of sphingolipids in cancer cells and discuss new avenues for future research at the intersection between oestrogen and sphingolipid signalling." @default.
- W2059700757 created "2016-06-24" @default.
- W2059700757 creator A5016117159 @default.
- W2059700757 creator A5056967574 @default.
- W2059700757 date "2013-12-09" @default.
- W2059700757 modified "2023-09-23" @default.
- W2059700757 title "Role of sphingolipids in oestrogen signalling in breast cancer cells: an update" @default.
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- W2059700757 doi "https://doi.org/10.1530/joe-13-0388" @default.
- W2059700757 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/24323911" @default.
- W2059700757 hasPublicationYear "2013" @default.
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