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• W2059727588 abstract "Abstract The current study examined the role of injured human kidney tubular epithelial cell (HKC) in the mediation of formation of calcium oxalate (CaOxa) crystals by means of scanning electronic microscopy and X-ray diffraction. HKC was injured using different concentrations of H2O2. Cell injury resulted in a significant decrease in cell viability and superoxide dismutase (SOD) concentration and an increase in the level of malondialdehyde (MDA) and expression of osteopontin (OPN). Injured cells not only promote nucleation and aggregation of CaOxa crystals, but also induce the formation of calcium oxalate monohydrate (COM) crystals that strongly adhere to cells. These results imply that injured HKCs promote stone formation by providing more nucleating sites for crystals, promoting the aggregation of crystals, and inducing the formation of COM crystals." @default.
• W2059727588 created "2016-06-24" @default.
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• W2059727588 date "2012-05-01" @default.
• W2059727588 modified "2023-09-23" @default.
• W2059727588 title "Mediation of calcium oxalate crystal growth on human kidney epithelial cells with different degrees of injury" @default.
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• W2059727588 doi "https://doi.org/10.1016/j.msec.2012.01.035" @default.
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