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- W2060073882 abstract "There is increasing interest in endocytosis that occurs independently of clathrin coats and the fates of membrane proteins internalized by this mechanism. The appearance of clathrin-independent endocytic and membrane recycling pathways seems to vary with different cell types and cargo molecules. In this review we focus on studies that have been performed using HeLa and COS cells as model systems for understanding this membrane trafficking system. These endosomal membranes contain signaling molecules including H-Ras, Rac1, Arf6 and Rab proteins, and a lipid environment rich in cholesterol and PIP2 providing a unique platform for cell signaling. Furthermore, activation of some of these signaling molecules (H-Ras, Rac and Arf6) can switch the constitutive form of clathrin-independent endocytosis into a stimulated one, associated with PM ruffling and macropinocytosis." @default.
- W2060073882 created "2016-06-24" @default.
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- W2060073882 creator A5049577856 @default.
- W2060073882 creator A5064758340 @default.
- W2060073882 date "2009-01-01" @default.
- W2060073882 modified "2023-10-07" @default.
- W2060073882 title "Clathrin-independent endocytosis: A unique platform for cell signaling and PM remodeling" @default.
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- W2060073882 doi "https://doi.org/10.1016/j.cellsig.2008.06.020" @default.
- W2060073882 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/2754696" @default.
- W2060073882 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/18647649" @default.
- W2060073882 hasPublicationYear "2009" @default.
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