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- W2060518035 abstract "Latent Epstein–Barr virus (EBV) infection is strongly associated with B-cell proliferative diseases such as Burkitt's lymphoma. Here we show that the oncogenic serine/threonine kinases Pim-1 and Pim-2 enhance the activity of the viral transcriptional activator EBNA2. During EBV infection of primary B-lymphocytes, the mRNA expression levels of pim genes, especially of pim-2, are upregulated and remain elevated in latently infected B-cell lines. Thus, EBV-induced upregulation of Pim kinases and Pim-stimulated EBNA2 transcriptional activity may contribute to the ability of EBV to immortalize B-cells and predispose them to malignant growth." @default.
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- W2060518035 date "2005-03-01" @default.
- W2060518035 modified "2023-10-07" @default.
- W2060518035 title "Pim kinases are upregulated during Epstein–Barr virus infection and enhance EBNA2 activity" @default.
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- W2060518035 doi "https://doi.org/10.1016/j.virol.2005.01.001" @default.
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