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• W2060998763 abstract "Epstein-Barr virus (EBV) is the causal agent in the etiology of Burkitt’s lymphoma and nasopharyngeal carcinoma and is also associated with multiple human malignancies, including Hodgkin’s and non-Hodgkin’s lymphoma, and posttransplantation lymphoproliferative disease, as well as sporadic cancers of other tissues. A causal relationship of EBV to these latter malignancies remains controversial, although the episomic EBV genome in most of these cancers is clonal, suggesting infection very early in the development of the tumor and a possible role for EBV in the genesis of these diseases. Furthermore, the prognosis of these tumors is invariably poor when EBV is present, compared to their EBV-negative counterparts. The physical presence of EBV in these tumors represents a potential “tumor-specific” target for therapeutic approaches. While treatment options for other types of herpesvirus infections have evolved and improved over the last two decades, however, therapies directed at EBV have lagged. A major constraint to pharmacological intervention is the shift from lytic infection to a latent pattern of gene expression, which persists in those tumors associated with the virus. In this paper we provide a brief account of new virus-targeted therapeutic approaches against EBV-associated malignancies." @default.
• W2060998763 created "2016-06-24" @default.
• W2060998763 creator A5076026191 @default.
• W2060998763 creator A5083735394 @default.
• W2060998763 creator A5086539163 @default.
• W2060998763 date "2012-01-01" @default.
• W2060998763 modified "2023-10-07" @default.
• W2060998763 title "Advances in Virus-Directed Therapeutics against Epstein-Barr Virus-Associated Malignancies" @default.
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• W2060998763 doi "https://doi.org/10.1155/2012/509296" @default.
• W2060998763 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/3303631" @default.
• W2060998763 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/22500168" @default.
• W2060998763 hasPublicationYear "2012" @default.
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