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- W2061036776 abstract "The tight junction (TJ) is a critical cellular component for maintenance of tissue integrity, cellular interactions and cell-cell communications, and physiologically functions as the great wall against external agents and the surrounding hostile environment. During the host-pathogen evolution, viruses somehow found the key to unlock the gate for their entry into cells and to exploit and exhaust the host cells. In the liver, an array of TJ molecules is localized along the bile canaliculi forming the blood-biliary barrier, where they play pivotal roles in paracellular permeability, bile secretion, and cell polarity. In pathology, certain hepatic TJ molecules mediate virus entry causing hepatitis infection; deregulation and functional abnormality of the TJ have also been implicated in triggering liver cancer development and metastasis. All these findings shed new insights on the understanding of hepatic TJs in the development of liver disease and provide new clues for potential intervention." @default.
- W2061036776 created "2016-06-24" @default.
- W2061036776 creator A5045317767 @default.
- W2061036776 creator A5089804910 @default.
- W2061036776 date "2010-01-01" @default.
- W2061036776 modified "2023-10-17" @default.
- W2061036776 title "Hepatic tight junctions: From viral entry to cancer metastasis" @default.
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- W2061036776 doi "https://doi.org/10.3748/wjg.v16.i3.289" @default.
- W2061036776 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/2807947" @default.
- W2061036776 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/20082472" @default.
- W2061036776 hasPublicationYear "2010" @default.
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