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- W2061748610 endingPage "212" @default.
- W2061748610 startingPage "203" @default.
- W2061748610 abstract "© Many viruses and bacteria have been identified as affecting atherosclerosis plaque deposition among them, dental and other oral pathogens (discussed in Part I of this series). Chlamydia pneumoniae probably has the strongest association with atherosclerosis.1-43 Other major atherosclerosis-associated pathogens are H. pylori and CMV, which by secreting vacuolating cytotoxin (H. pylori)44-54 or by inducing proliferation of smooth-muscle cells (CMV) (discussed in Part III of this series) may accelerate atherosclerosis formation. Infectious agents can also contribute to the acceleration of atherosclerosis development; either by nonspecific mechanisms, such as hypercoagulation, increased production of adhesion molecules, and elevated C-reactive protein (CRP) levels (discussed in Part I of this series), or they can act by more specific mechanisms, such as induction of HSP60 expression and eventually pathogenic anti-HSP-60 antibody production (discussed in Part III of this series)." @default.
- W2061748610 created "2016-06-24" @default.
- W2061748610 creator A5017537703 @default.
- W2061748610 date "2003-03-01" @default.
- W2061748610 modified "2023-09-27" @default.
- W2061748610 title "A Look at Infectious Agents as a Possible Causative Factor in Cardiovascular Disease: Part I" @default.
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