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• W2061864654 abstract "In vivo administration of thyroxine (T4) to CBA/J, C3H/HeN, and B6C3F1 mice resulted in enhanced splenic and peritoneal natural killer (NK) cell activity against YAC-1 lymphoma target cells in vitro. T4 treatment did not augment splenic or peritoneal NK activity in A/J, BALB/c, C57BL/6, or DBA/2 mice. Natural killer cells from T4-treated mice did not lyse P815 cells and were not removed by treatment with anti-Thy 1.2 + complement. Effector cell binding to target cells was not altered following T4 treatment. CBA/J mice treated with T4 also exhibit enhanced in vivo NK activity compared with untreated mice as demonstrated by rapid clearance of [125I]iodo-2′-deoxyuridine-labeled YAC-1 lymphoma cells. The augmentation of NK activity as a result of T4 treatment is reversible and is not accompanied by increased serum interferon levels. Spleen cells from T4-treated mice incorporate more [3H]thymidine and [3H]uridine in comparison to spleen cells from control animals. Treatment of mice with 131I did not enhance splenic or peritoneal NK activity. These data suggest that thyroid hormones can augment mouse NK activity and this modulation is most likely due to changes in the metabolic properties of NK cells." @default.
• W2061864654 created "2016-06-24" @default.
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• W2061864654 date "1982-10-01" @default.
• W2061864654 modified "2023-09-26" @default.
• W2061864654 title "Enhancement of mouse natural killer cell activity by thyroxine" @default.
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• W2061864654 doi "https://doi.org/10.1016/0008-8749(82)90437-3" @default.
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