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- W2061940897 abstract "Purpose: Despite major advances in the treatment of cardiovascular diseases, heart failure (HF) remains one of the top causes of death world-wide. The implications of microRNAs in this process are well accepted but still only incompletely understood. The microRNAs 221-3p and 222-3p are processed from a common precursor and share the same seed sequence and thus form the microRNA-221/222 family (miR-221/222). Both microRNAs were found to be involved in myoblast differentiation and are upregulated after aortic banding in mice. We therefore hypothesized that the miR-221/222 family is involved in the pathophysiology of cardiac hypertrophy and failure upon pressure overload." @default.
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- W2061940897 date "2014-06-27" @default.
- W2061940897 modified "2023-10-16" @default.
- W2061940897 title "P69The microRNA-221/222 family is differentially regulated in cardiac disease and counteracts pressure overload-induced cardiac remodeling in mice" @default.
- W2061940897 doi "https://doi.org/10.1093/cvr/cvu082.13" @default.
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