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- W2062007901 abstract "A number of ion mechanisms are involved in the stimulus-secretion coupling in the pancreatic 8-cells. We have previously produced evidence that transmembraneC1-fluxes may be important for normal insulin release (11, 12) and that disturbed C1fluxes may be connected with the deterioration of insulin release in diabetic mouse 8-cells (2). The C1mechanisms operating in mouse pancreatic islets appear to consist of both a metabolism-dependent inward transport of C1against its electrochemical potential gradient and an efflux controlled by the islet cell C1permeability and the magnitude of an outwardly directed electrochemical potential gradient (11 , 12). The work presented here tested further the possible relationship between transmembrane C1fluxes and insulin release by using either insulin secretagogues, i.e. L-leucine, glibenclamide or theophylline or the C1transport inhibitor furosemide." @default.
- W2062007901 created "2016-06-24" @default.
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- W2062007901 date "1981-01-01" @default.
- W2062007901 modified "2023-09-25" @default.
- W2062007901 title "Are Cl<sup>−</sup>Mechanisms in Mouse Pancreatic Islets involved in Insulin Release?" @default.
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- W2062007901 doi "https://doi.org/10.3109/03009738109179226" @default.
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