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- W2062572018 abstract "# {#article-title-2} To the Editor: Shimizu et al.1 report increased demyelination and inflammation following interferon-β-1b (IFNβ-1b) therapy in patients with opticospinal multiple sclerosis (OSMS) and neuromyelitis optica (NMO). In the accompanying editorial, Drs. Weinshenker and Wingerchuk2 discuss whether a shift of immune response secondary to treatment with IFNβ-1b may account for the unpredictable response. We contend that the offset of immune response capable of unpredictable demyelinating attacks is unknown and not necessarily confined to B-cell activity or Th2 shift.A 42-year-old Caucasian woman had, over the course of 6 months, 2 episodes of optic neuritis—one in each eye—and 2 attacks to the spinal cord consistent with acute recurrent myelitis. The MRI brain showed no signs of demyelination and, despite negative NMO–immunoglobulin G (IgG), the patient was started on rituximab. Within 2 weeks, she developed a left hemispheric brain lesion similar to the one shown in figure 1A.1The brain lesion continued to expand and after 6 courses of monthly cyclophosphamide the active process was suppressed. The pathology obtained via brain biopsy revealed reactive hypercellularity with …" @default.
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- W2062572018 date "2011-07-12" @default.
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- W2062572018 title "IFN -1b may severely exacerbate Japanese opticspinal MS in neuromyelitis optica spectrum: Japanese optic-spinal MS: Is it MS or neuromyelitis optica and does the answer dictate treatment?" @default.
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- W2062572018 doi "https://doi.org/10.1212/wnl.0b013e318219dde5" @default.
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