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- W2063291275 abstract "To the Editor.— The reader who is not highly familiar with the recent developments concerning vitamin D and its various active forms may be misled by certain comments made (235: 164, 1976) by Teitelbaum and associates. First, the major reason for the interest in the treatment of renal osteodystrophy with 1,25-dihydroxycholecalciferol (1,25-dihydroxy-vitamin D 3 [1,25 {OH} 2 D 3 ]) and 1α-hydroxycholecalciferol (1α-hydroxy-vitamin D 3 [1α {OH} D 3 ]) arises not because of antirachitic properties of vitamin D in uremia, but because of discoveries that these compounds bypass the necessity for 1-hydroxylation in the kidney. 1,2 Thus, the kidney is the only known organ capable of converting 25(OH)D 3 to 1,25(OH) 2 D 3 , the most active, naturally occurring form of vitamin D; also, 1,25(OH) 2 D 3 may account for all biologic actions heretofore ascribed to vitamin D itself. Moreover, a characteristic of renal osteodystrophy is a resistance to treatment" @default.
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- W2063291275 date "1976-07-26" @default.
- W2063291275 modified "2023-09-26" @default.
- W2063291275 title "Calcifediol in Chronic Renal Insufficiency" @default.
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- W2063291275 doi "https://doi.org/10.1001/jama.1976.03270040013015" @default.
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