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- W2063359467 abstract "Abnormalities in thirst and vasopressin (AVP) release play key roles in the genesis of hyponatremia; both processes are under the control of osmoreceptive neurons in the central nervous system (CNS). The acute development of hyponatremia in turn leads to profound cerebral edema, whereas treatment of chronic hyponatremia can be associated with osmotic demyelination syndrome (ODS). The brain is thus both culprit and victim in hyponatremia. This review summarizes recent advances in the understanding of osmoreception in the brain, the CNS response to acute and chronic hyponatremia, and the pathophysiology of ODS." @default.
- W2063359467 created "2016-06-24" @default.
- W2063359467 creator A5074145664 @default.
- W2063359467 date "2009-05-01" @default.
- W2063359467 modified "2023-09-24" @default.
- W2063359467 title "The Brain in Hyponatremia: Both Culprit and Victim" @default.
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