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- W2064139379 endingPage "1042" @default.
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- W2064139379 abstract "TNF-α, a pro-inflammatory cytokine, exerts its biological activity by signaling via its two receptors, TNFR-1 and TNFR-2 and by activating NF-κB. NF-κB is essential for survival of many cell types; however, TNF-α also induces cell death. During human aging TNF-α production and TNF-α-induced apoptosis are increased. In this brief review, we will discuss the TNF-α-mediated signaling and various molecular mechanisms underlying increased apoptosis in T cell subsets in aged humans with a special reference to alterations in transcription factor NF-κB and Fas-associated death domain (FADD)." @default.
- W2064139379 created "2016-06-24" @default.
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- W2064139379 date "2005-05-01" @default.
- W2064139379 modified "2023-09-24" @default.
- W2064139379 title "Molecular mechanisms of TNF-α-induced apoptosis in aging human T cell subsets" @default.
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- W2064139379 doi "https://doi.org/10.1016/j.biocel.2004.08.013" @default.
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