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• W2064156794 abstract "Purpose of review This paper reviews recent molecular genetic studies of schizophrenia and evaluates claims implicating specific genes as susceptibility loci. Recent findings Molecular genetic studies have identified several potential regions of linkage and two associated chromosomal abnormalities, and the evidence is accumulating in favour of several positional candidate genes. Currently, the strongest evidence for putative schizophrenia susceptibility loci relates to the genes encoding dysbindin (DTNBP1) and neuregulin (NRG1). For other genes, disrupted in schizophrenia (DISC1), D-amino acid oxidase activator (DAOA), regulator of G-protein signalling 4 (RGS4) and V-AKT murine thymoma viral oncogene homolog 1 (AKT1) the data are promising but not yet compelling. In the most convincing cases, the risk haplotypes appear to be associated with small effect sizes and do not fully explain the linkage findings that prompted each study. Summary The ability of positional genetics to implicate novel genes and pathways will open up new vistas for neurobiological research. Despite the accumulation of significant genetic data, however, the susceptibility variants have yet to be identified and detailed follow-up studies are now required." @default.
• W2064156794 created "2016-06-24" @default.
• W2064156794 creator A5009170632 @default.
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• W2064156794 date "2006-03-01" @default.
• W2064156794 modified "2023-10-17" @default.
• W2064156794 title "An update on the genetics of schizophrenia" @default.
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• W2064156794 doi "https://doi.org/10.1097/01.yco.0000214341.52249.59" @default.
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