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• W2064354792 endingPage "148" @default.
• W2064354792 startingPage "142" @default.
• W2064354792 abstract "To study the mechanism of the L-type voltage-gated calcium channel (L-VGCC) involved in neuronal injury induced by brain ischemia and reperfusion (I/R), transient (15 min) brain ischemia was induced by four-vessel occlusion of Sprague-Dawley (SD) rats. Tyrosine phosphorylation of NR2A and interaction of NR2A with Src and Pyk2 in hippocampus induced by brain ischemia and reperfusion (I/R) were determined by immunoprecipitation and immunoblot(ting). Tyrosine phosphorylation of NR2A in hippocampus was enhanced after I/R. Interaction of NR2A with Src and Pyk2, tyrosine phosphorylation and kinase activity of Src and Pyk2 also increased after I/R. All the increases were partly inhibited by L-VGCC antagonist nifedipine administered to rats 20 min prior to brain ischemia. The results suggested that increase of tyrosine phosphorylation of NR2A induced by I/R had a relation to activation of L-VGCC. Src and Pyk2 interacting with NR2A might also be involved in this regulation of the tyrosine phosphorylation of NR2A induced by I/R." @default.
• W2064354792 created "2016-06-24" @default.
• W2064354792 creator A5013233801 @default.
• W2064354792 creator A5033080900 @default.
• W2064354792 creator A5065816927 @default.
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• W2064354792 date "2003-05-01" @default.
• W2064354792 modified "2023-09-23" @default.
• W2064354792 title "L-type voltage-gated calcium channel attends regulation of tyrosine phosphorylation of NMDA receptor subunit 2A induced by transient brain ischemia" @default.
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• W2064354792 doi "https://doi.org/10.1016/s0006-8993(03)02519-8" @default.
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• W2064354792 hasPublicationYear "2003" @default.
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