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• W2064470478 abstract "Myeloid cells are key effectors of the innate immune response, and as such are often required to migrate to, and function within, sites that are markedly hypoxic. To adapt to such oxygen deplete environments they have developed functional and survival responses that are regulated by the hypoxia-inducible factor (HIF) oxygen-sensing pathway. In this review, we describe three key aspects of HIF-dependent regulation of myeloid cell function: (i) the maintenance of ATP pools and the subsequent regulation of proinflammatory responses, (ii) the HIF-dependent inhibition of neutrophil apoptosis and (iii) the HIF-mediated regulation β2 integrin expression. Myeloid cells are key effectors of the innate immune response, and as such are often required to migrate to, and function within, sites that are markedly hypoxic. To adapt to such oxygen deplete environments they have developed functional and survival responses that are regulated by the hypoxia-inducible factor (HIF) oxygen-sensing pathway. In this review, we describe three key aspects of HIF-dependent regulation of myeloid cell function: (i) the maintenance of ATP pools and the subsequent regulation of proinflammatory responses, (ii) the HIF-dependent inhibition of neutrophil apoptosis and (iii) the HIF-mediated regulation β2 integrin expression." @default.
• W2064470478 created "2016-06-24" @default.
• W2064470478 creator A5012684804 @default.
• W2064470478 creator A5048100194 @default.
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• W2064470478 date "2005-08-01" @default.
• W2064470478 modified "2023-09-27" @default.
• W2064470478 title "The role of HIF-1α in myeloid cell inflammation" @default.
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• W2064470478 doi "https://doi.org/10.1016/j.it.2005.06.007" @default.
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