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- W2064874908 abstract "HYPOGLYCEMIA in the neonate or young infant is recognized to be a metabolic error with potentially devastating effects on the brain. Neurologic sequelae may occur in the adult, but susceptibility to hypoglycemia is much greater in the immature, poorly myelinated brain of the infant. In addition, the general metabolic immaturity of the neonate allows disturbances of carbohydrate metabolism in a variety of circumstances, such as cold exposure, 1 congestive heart failure, 2 or idiopathic respiratory distress of the newborn. 3 Hypoglycemia in the young infant may be defined as true blood glucose level less than 20 mg/100 ml in the premature infant; less than 30 mg/100 ml in the full-term infant in the first 48 hours of life; and less than 40 mg/100 ml thereafter. 4 Manifestations suggestive of hypoglycemia include pallor, lethargy, limpness, absent Moro reflex, rolling of the eyes or the abrupt appearance of internal strabismus," @default.
- W2064874908 created "2016-06-24" @default.
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- W2064874908 date "1970-10-01" @default.
- W2064874908 modified "2023-10-16" @default.
- W2064874908 title "Hypoglycemia Due to Organic Hyperinsulinism in Infancy" @default.
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- W2064874908 doi "https://doi.org/10.1001/archneur.1970.00480280044005" @default.
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