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• W2065209043 abstract "Hybridoma cell lines producing natural autoantibodies (NAA), generated from A.SW mice with progressive experimental autoimmune encephalomyelitis (P-EAE), have been shown to cause demyelination and renal pathology when injected into naïve mice. To investigate the relative contribution of these antibodies to disease pathogenesis, B-1 cells, the major producers of NAA, were depleted by hypotonic shock. Depletion of B-1 cells during the effector phase of EAE significantly decreased the severity of demyelination and overall pathology in the brain. There was also a decreased incidence of P-EAE and a decrease in clinical score. Depletion during the induction phase of the disease resulted in an increase in the incidence of P-EAE and in the clinical score. Overall, B-1 cells were found to modulate EAE pathogenesis." @default.
• W2065209043 created "2016-06-24" @default.
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• W2065209043 date "2008-01-01" @default.
• W2065209043 modified "2023-09-25" @default.
• W2065209043 title "Role of CD5⁺B-1 cells in EAE pathogenesis" @default.
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• W2065209043 doi "https://doi.org/10.1080/08916930801890280" @default.
• W2065209043 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/4399970" @default.
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