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• W2065388184 abstract "The etiology of aphakic cystoid macular edema (ACME) is still unclear. It is associated with many conditions and probably represents a nonspecific response of the macula to a host of stimuli under the circumstances of altered stability of the vitreous cavity secondary to aphakia. Recognizing the alteration in the vitreous body and its relationship to intraocular structures in aphakia is important in understanding the milieu in which ACME occurs. Aphakic vitreous changes in the posterior vitreous surface, including posterior vitreous detachment and vitreomacular traction; anterior vitreous changes, including rupture of the anterior hyaloid face and incarceration of vitreous in the corneal wound, and changes in the vitreous gel, including vitreous collapse and loss of hyaluronic acid, are reviewed in light of their relationship with ACME. The theoretical merits of surgical approach aimed at producing the least amount of structural and physiologic changes in the intraocular structures from phakia to aphakia are also discussed." @default.
• W2065388184 created "2016-06-24" @default.
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• W2065388184 date "1982-06-01" @default.
• W2065388184 modified "2023-10-18" @default.
• W2065388184 title "The role of vitreous in aphakic cystoid macular edema: A review" @default.
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• W2065388184 doi "https://doi.org/10.1016/s0146-2776(82)80081-5" @default.
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