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- W2065721859 startingPage "235" @default.
- W2065721859 abstract "Epstein–Barr virus (EBV), a B‐lymphotropic gamma‐herpesvirus, causes infectious mononucleosis and oral hairy leukoplakia, and is associated with various types of lymphoid and epithelial malignancies. Saliva is the main vehicle for EBV transmission from individual to individual. Recent studies have also implicated EBV in the pathogenesis of advanced types of periodontal disease. EBV DNA is detected in 60–80% of aggressive periodontitis lesions and in 15–20% of gingivitis lesions or normal periodontal sites. The periodontal presence of EBV is associated with an elevated occurrence of periodontopathic anaerobic bacteria. Moreover, EBV active infection occurs in ≈ 70% of symptomatic and large‐size periapical lesions. EBV and cytomegalovirus often co‐exist in marginal and apical periodontitis. Periodontal therapy can markedly suppress the EBV load in periodontal pockets as well as in saliva, which has the potential to reduce the risk of viral transmission between close individuals. EBV proteins up‐regulate cytokines and growth factors, which seem to play a central role in the proliferative response of tongue epithelial cells in oral hairy leukoplakia and in the cell‐transformation process of EBV‐associated malignancies. Further research is needed to identify the full range of EBV‐related diseases in the human oral cavity." @default.
- W2065721859 created "2016-06-24" @default.
- W2065721859 creator A5028143719 @default.
- W2065721859 creator A5033236089 @default.
- W2065721859 creator A5049460096 @default.
- W2065721859 creator A5077889251 @default.
- W2065721859 date "2006-06-26" @default.
- W2065721859 modified "2023-10-17" @default.
- W2065721859 title "Epstein-Barr virus in oral diseases" @default.
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