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- W2065754675 endingPage "618" @default.
- W2065754675 startingPage "611" @default.
- W2065754675 abstract "The advent of the selective cholinergic toxin, 192 IgG-saporin, dramatically shaped subsequent research on the role of the basal forebrain in learning and memory. In particular, several articles (including the authors' 1995 Behavioral Neuroscience paper; M. G. Baxter, D. J. Bucci, L. K., Gorman, R. G. Wiley, & M. Gallagher, 1995) revealed that selective removal of basal forebrain cholinergic neurons had surprisingly little effect on spatial learning and memory. Here, as part of the series commemorating the 30th anniversary of Behavioral Neuroscience, we describe how our earlier findings prompted a reconsideration of the cholinergic contribution to cognitive function and also led to several new research directions, including renewed interest in basal forebrain GABA-ergic neurons and cholinergic contributions to neurocognitive development. The authors also describe how the successful use of 192 IgG-saporin led to the development and popularity of a wide range of selective new neurotoxic agents. Finally, they consider the utility of the permanent lesion approach in the wake of new transgenic and optogenetic methods." @default.
- W2065754675 created "2016-06-24" @default.
- W2065754675 creator A5025402084 @default.
- W2065754675 creator A5091356159 @default.
- W2065754675 date "2013-01-01" @default.
- W2065754675 modified "2023-10-17" @default.
- W2065754675 title "Selective immunotoxic lesions of basal forebrain cholinergic neurons: Twenty years of research and new directions." @default.
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