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- W2065822015 abstract "A new hypothesis is presented for the first time to explain the etiology of osteoporosis. Prostaglandins (E2 and F2 alpha) at precise concentrations, have been observed to be involved in bone formation. A close association exists between levels of prostaglandins (E2 and F2 alpha) demonstrated in the neonatal mouse leading to bone formation, with estimated prostaglandins (E2 and F2 alpha) concentrations reported in man. Several hormones (vasopressin, oxytocin, luteinizing hormone, follicle-stimulating hormone, cortisol, estradiol, and testosterone) can indirectly affect prostaglandin formation leading to reduced bone formation. The association between these hormones and prostaglandins (E2 and F2 alpha) explains the physiological mechanism whereby estradiol can be effective for the treatment of osteoporosis. This association also explains the etiology of lumbar spondylitis/spondylodynia, reasons for complaints of increased pain in wet cold weather among arthritics and a multitude of other events. Mechanisms related to this interaction between various hormones and the effect of prostaglandins (E2 and F2 alpha) on bone formation are discussed." @default.
- W2065822015 created "2016-06-24" @default.
- W2065822015 creator A5086032375 @default.
- W2065822015 date "1992-06-01" @default.
- W2065822015 modified "2023-09-25" @default.
- W2065822015 title "New clues into the etiology of osteoporosis: The effects of prostaglandins (E2 and F2α) on bone" @default.
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- W2065822015 doi "https://doi.org/10.1016/0306-9877(92)90085-q" @default.
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