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• W2066356532 startingPage "1260" @default.
• W2066356532 abstract "Injury elicits a response from all cells of the immune system in which cytokines and other metabolic products of activated leukocytes can act either beneficially to provide for enhanced host resistance or deleteriously to depress the function of remote organs and cause what has been termed<i>systemic inflammation</i>. These at times antithecal responses of leukocytes that appear to integrate postinjury changes in the neuroendocrine, immune, and coagulation systems have been implicated as principal causative factors in multiple systems organ failure. Numerous investigators have evaluated a variety of therapeutic agents to prevent and control infection by restoring leukocyte function, while others have evaluated antagonists and monoclonal antibodies as a means of controlling the exaggerated and persistent actions of leukocytes and cytokines caused by systemic inflammation. The redundancies of the cell populations and the cytokines and other metabolites produced by the cells predictably limit the effectiveness of any single agent and make clinical evaluation of such agents difficult. (Arch Surg. 1993;128:1260-1267)" @default.
• W2066356532 created "2016-06-24" @default.
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• W2066356532 creator A5082149162 @default.
• W2066356532 date "1993-11-01" @default.
• W2066356532 modified "2023-09-27" @default.
• W2066356532 title "Leukocyte Responses to Injury" @default.
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• W2066356532 doi "https://doi.org/10.1001/archsurg.1993.01420230088014" @default.
• W2066356532 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/8239989" @default.
• W2066356532 hasPublicationYear "1993" @default.
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