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- W2066690639 abstract "The aim of the study was to determine whether the presence of angiotensin II type 1 receptor 1166A/C gene polymorphism and two polymorphisms of angiotensinogen, namely Met235Thr and Thr174Met, pointed at the culprit artery in patients with ST-segment elevation myocardial infarction (STEMI). The AGTR1 1166A/C polymorphism and two AGT gene polymorphisms, Met235Thr and Thr174Met, were assessed in 100 patients with STEMI. The odds ratio (OR) of circumflex artery (CRX) responsible for STEMI was 3.49 (95% CI: 1.1–10.8, p < 0.05) for MetThr genotype for AGT Met235Thr gene and 4.54 (95% CI: 1.5–14.2, p < 0.01) for ThrMet genotype for AGT Thr174Met gene. Homozygous ThrThr genotype for AGT Thr174Met gene reduced OR of CRX as culprit artery in STEMI (OR = 0.29, 95% CI: 0.1–0.9, p < 0.05). However, the highest OR that increased up to 13.71 (95% CI: 1.58–119.3) was shown in case of right coronary artery and C/C genotype for AGTR1 1166A/C gene. Polymorphism of AGTR1 1166A/C gene can point at the right coronary artery as infarct-related artery in STEMI. Polymorphisms of AGT Met235Thr and AGT Thr174Met genes are able to mark increased or reduced odds ratio of circumflex artery as culprit artery in STEMI." @default.
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- W2066690639 date "2012-05-01" @default.
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- W2066690639 title "Polymorphisms of angiotensin II type 1 receptor gene and those of angiotensinogen point at culprit artery in ST-segment elevation myocardial infarction" @default.
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- W2066690639 doi "https://doi.org/10.1016/j.gene.2012.03.014" @default.
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