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- W2066900682 abstract "Long-term (14 days) treatment of mice with the antidepressant drug imipramine increased the number of GABA-B receptors in the cerebral cortex and also led to an increase in the potentiation of norepinephrine-induced cAMP accumulation by baclofen (a GABA-B agonist) in cortical slices. Chronic baclofen treatment decreased both of these measures. These results suggest that previous evidence of increased GABA-B binding by antidepressants is coupled to a functional increase in adenylate cyclase activity, but that the mechanism responsible for this effect is not due simply to direct GABA-B receptor stimulation." @default.
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- W2066900682 date "1986-11-01" @default.
- W2066900682 modified "2023-09-25" @default.
- W2066900682 title "Effect of chronic imipramine or baclofen on GABA-B binding and cyclic AMP production in cerebral cortex" @default.
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- W2066900682 doi "https://doi.org/10.1016/0014-2999(86)90526-1" @default.
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