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- W2067690802 abstract "Prolactin (PRL) activates PRL receptor isoforms to exert regulation of specific neuronal circuitries, and to control numerous physiological and clinically-relevant functions including; maternal behavior, energy balance and food intake, stress and trauma responses, anxiety, neurogenesis, migraine and pain. PRL controls these critical functions by regulating receptor potential thresholds, neuronal excitability and/or neurotransmission efficiency. PRL also influences neuronal functions via activation of certain neurons, resulting in Ca2+ influx and/or electrical firing with subsequent release of neurotransmitters. Although PRL was identified almost a century ago, very little specific information is known about how PRL regulates neuronal functions. Nevertheless, important initial steps have recently been made including the identification of PRL-induced transient signaling pathways in neurons and the modulation of neuronal transient receptor potential (TRP) and Ca2+-dependent K+ channels by PRL. In this review, we summarize current knowledge and recent progress in understanding the regulation of neuronal excitability and channels by PRL." @default.
- W2067690802 created "2016-06-24" @default.
- W2067690802 creator A5002121997 @default.
- W2067690802 creator A5033043600 @default.
- W2067690802 creator A5070489124 @default.
- W2067690802 date "2014-04-23" @default.
- W2067690802 modified "2023-10-18" @default.
- W2067690802 title "Prolactin receptor in regulation of neuronal excitability and channels" @default.
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- W2067690802 doi "https://doi.org/10.4161/chan.28946" @default.
- W2067690802 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/4203747" @default.
- W2067690802 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/24758841" @default.