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- W2068442057 abstract "Emerging evidence indicates that CD4(+) T cells possess cytotoxic potential for tumor eradication and perforin/granzyme-mediated cytotoxicity functions as one of the important mechanisms for CD4(+) T cell-triggered cell killing. However, the critical issue is how the cytotoxic CD4(+) T cells are developed. During the course of our work that aims at promoting immunostimulation of APCs by inhibition of negative regulators, we found that A20-silenced Mф drastically induced granzyme B expression in CD4(+) T cells. As a consequence, the granzyme-highly expressing CD4(+) T cells exhibited a strong cytotoxic activity that restricted tumor development. We found that A20-silenced Mф activated cytotoxic CD4(+) T cells by MHC class-II restricted mechanism and the activation was largely dependent on enhanced production of IFN-γ." @default.
- W2068442057 created "2016-06-24" @default.
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- W2068442057 date "2012-11-07" @default.
- W2068442057 modified "2023-09-24" @default.
- W2068442057 title "A20 Controls Macrophage to Elicit Potent Cytotoxic CD4+ T Cell Response" @default.
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- W2068442057 doi "https://doi.org/10.1371/journal.pone.0048930" @default.
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