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• W2068751406 endingPage "645" @default.
• W2068751406 startingPage "641" @default.
• W2068751406 abstract "Duchenne's muscular dystrophy (DMD), which affects 1/3500 live male births, involves a progressive degeneration of skeletal and cardiac muscle, leading to early death. The protein dystrophin is lacking in DMD and present, but defective, in the allelic, less severe, Becker muscular dystrophy and is also missing in the mdx mouse. Experiments on the mdx mouse have suggested two possible therapies for these myopathies. Implantation of normal muscle precursor cells (mpc) into mdx skeletal muscle leads to the conversion of dystrophin-negative fibres to -positive, with consequent improvement in muscle histology. Direct injectidn of dystrophin cDNA into skeletal or cardiac muscle also gives rise to dystrophin-positive fibres. Although both appear promising, there are a number of questions to be answered and refinements to be made before either technique could be considered possible as treatments for myopathies in man." @default.
• W2068751406 created "2016-06-24" @default.
• W2068751406 creator A5046261021 @default.
• W2068751406 creator A5055891535 @default.
• W2068751406 date "1992-09-01" @default.
• W2068751406 modified "2023-10-16" @default.
• W2068751406 title "Challenges: Cell transplantation and gene therapy in muscular dystrophy" @default.
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• W2068751406 doi "https://doi.org/10.1002/bies.950140913" @default.
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• W2068751406 hasPublicationYear "1992" @default.
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