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- W2069290513 abstract "A decrease in the extracellular concentration of K+ provokes a dose-related, progressive, and persistent inhibition of insulin release evoked by high concentrations of glucose or alpha-ketoisocaproate. The biosynthesis of proinsulin and other peptides is severely decreased in the K+-deprived islets. The functional situation found in the K+-deprived islets can be mimicked, to a limited extent, by exposing the islets to inhibitors of protein biosynthesis and is reminiscent of that found in glucose-deprived islets. It is postulated that the intracellular concentration of K+ in islet cells participates in the long term regulation of insulin release by glucose." @default.
- W2069290513 created "2016-06-24" @default.
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- W2069290513 date "1980-03-01" @default.
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- W2069290513 title "The Stimulus-Secretion Coupling of Glucose-Induced Insulin Release. XXXIX. Long Term Effects of K+ Deprivation upon Insulin Biosynthesis and Release*" @default.
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- W2069290513 doi "https://doi.org/10.1210/endo-106-3-778" @default.
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