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- W2069603973 abstract "Vigabatrin was licensed in Britain and the Republic of Ireland in 1989 and has proved to be a valuable drug for the treatment of epilepsy which is not satisfactorily controlled by other anti-epileptic drugs and for initial monotherapy in West’s Syndrome. However, since 1990, there have been reports of both symptomatic (Eke et al., 1997; Wilson and Brodie, 1997; Wong et al., 1997; Blackwell et al., 1997) and asymptomatic (Mackenzie and Klistorner, 1998) visual field defects in patients treated with this drug (usually in combination with other anti-epileptic drugs). The overall incidence, based on epidemiological studies is estimated to be 14.5/10,000 patients with epilepsy a year (Martinez and Noack, 1997). A specific pattern of bilateral concentric constriction of the visual field is seen which is predominantly nasal with temporal sparing (Eke et al., 1997; Harding et al., 1997; Mackenzie and Klistorner, 1998). In some cases, these defects improved after Vigabatrin was stopped, but in others it persisted (Eke et al., 1997; Wilson and Brodie, 1997). We report on a patient with learning disability who presented with diplopia and blurred vision, after five years of Vigabatrin treatment. Ophthalmic examination revealed retinal epithelial atrophy around the macular area, with deposits." @default.
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- W2069603973 date "2001-01-01" @default.
- W2069603973 modified "2023-09-27" @default.
- W2069603973 title "Vigabatrin Treatment and Visual Problems in a Patient with Learning Disability" @default.
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- W2069603973 doi "https://doi.org/10.1179/096979501799155684" @default.
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