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• W2070933263 abstract "We compared the basal and bradykinin (BK)-induced NO release and endothelium-derived hyperpolarizing factor-mediated function between coronary arteries and cardiac veins.Isolated coronary vessels (large arteries, cardiac veins as well as microvessels) were studied. An NO-specific electrode and a conventional intracellular glass microelectrode were used to directly measure NO released from endothelial cells and hyperpolarization of smooth muscle cells in conduit coronary vessels. The basal and BK-induced release of NO was 14.2 +/- 2.0 nmol/L and 237.1 +/- 27.2 nmol/L (n = 8) in the artery, significantly greater than in veins (8.0 +/- 1.1 nmol/L and 135.6 +/- 14.5 nmol/L, n = 8, P < 0.01). The BK-induced hyperpolarization was significantly reduced by N-nitro-L-arginine, indomethacin, and hemoglobin in both arteries and veins and was greater in the arteries. The EDHF-mediated relaxation was significantly higher in the arteries than in veins, greater in microveins than in large veins, and almost abolished by charybdotoxin and apamin.Both NO and EDHF are involved in the regulation of the vascular tone in the coronary arterial and venous systems but the amount of NO release and the EDHF-mediated relaxation and associated hyperpolarization are less significant in the vein than in the artery in the coronary system." @default.
• W2070933263 created "2016-06-24" @default.
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• W2070933263 date "2004-06-01" @default.
• W2070933263 modified "2023-09-25" @default.
• W2070933263 title "Different Role of Nitric Oxide and Endothelium-Derived Hyperpolarizing Factor in Endothelium-Dependent Hyperpolarization and Relaxation in Porcine Coronary Arterial and Venous System" @default.
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• W2070933263 doi "https://doi.org/10.1097/00005344-200406000-00014" @default.
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