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- W207138683 abstract "ABSTRACT Galanin (Gal) is expressed in lactotrophs, thyrotrophs, somatotrophs, gonadotrophs, and corticotrophs of the anterior pituitary (AP). Gal enhances prolactin (PRL) release from lactotrophs, and there is evidence of its involvement in the development of prolactinomas. Less clear is the role of Gal in the functional control of other AP cells. Gal-positive nerve fibers are distributed around pancreatic islets, and Gal was found to inhibit agonist-stimulated insulin release. Gal is expressed in the adrenal medulla, and it has been shown that Gal directly stimulates glucocorticoid secretion acting in a paracrine manner. Neurotensin (NT) expression has been detected in AP lactotrophs, thyrotrophs, and gonadotrophs. NT enhances PRL release from lactotrophs, although contrasting findings have been obtained on its effect on thyrotrophs and gonadotrophs. NT is expressed in the pancreas and adrenal medulla, and evidence indicates that NT modulates insulin release and inhibits mineralocorticoid secretion. Neuromedin (NM) B is mainly contained in AP thyrotrophs, where its expression is upregulated by thyroid hormones. NMB inhibits thyroid-stimulating hormone release from thyrotrophs, leading to the hypothesis that a central feedback mechanism is operative by which the thyroid gland dampens its own exceedingly high secretory activity. NMB and NMC are expressed in the adrenal medulla, but their effect on adrenal secretion is unclear. Conversely, NMU has been reported to raise adrenal steroid production through an indirect paracrine mechanism involving the adrenal medulla." @default.
- W207138683 created "2016-06-24" @default.
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- W207138683 date "2006-01-01" @default.
- W207138683 modified "2023-09-26" @default.
- W207138683 title "Galanin, Neurotensin, and Neuromedins in the Local Regulation of Endocrine Glands" @default.
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- W207138683 doi "https://doi.org/10.1016/b978-012369442-3/50124-0" @default.
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