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• W2072095725 abstract "HLA-G is a non-classical HLA class I molecule involved in immunotolerance. HLA-G protects the fetus from maternal immune recognition and promotes allograft acceptance and tumor escape. Its low polymorphism and primary function, which is not peptide presentation to T lymphocytes, led us to compare the signal transduced after interaction between HLA-G and its receptor to those of innate immunity receptors with their ligands. We investigated the role of HLA-G in the classical NF-κB pathway in natural killer (NK) cells, which is the major pathway activated by innate immunity receptors. In NK cells stimulated with HLA-G1-expressed cells, we demonstrate that HLA-G induces the phosphorylation and the degradation of IκBα leading to nuclear translocation of NF-κB. This effect is independent of the presence of ILT-2 receptors and is still observed using a peptide corresponding to the alpha-1 domain of HLA-G. All these data support an unsuspected role for HLA-G in innate immunity by activating classical NF-κB pathway in NK cells." @default.
• W2072095725 created "2016-06-24" @default.
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• W2072095725 date "2008-01-01" @default.
• W2072095725 modified "2023-10-12" @default.
• W2072095725 title "Role of HLA-G in innate immunity through direct activation of NF-κB in natural killer cells" @default.
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• W2072095725 doi "https://doi.org/10.1016/j.molimm.2007.06.160" @default.
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