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• W2072360147 abstract "Gram-positive bacterial infections of the central nervous system, such as meningitis, induce an extensive inflammatory response, which in turn may damage neurons. LTA (lipoteichoic acid) is a component of the Gram-positive bacterial cell wall that induces glial inflammatory activation in vitro and in vivo. It does so by binding to Toll-like receptor-2 on microglia and astrocytes, rapidly activating ERK1/2 (extracellular-signal-regulated kinase 1/2) and p38 MAPKs (mitogen-activated protein kinases), causing NF-κB (nuclear factor κB) activation and leading to the production of pro-inflammatory cytokines and expression of inducible nitric oxide synthase (in synergy with muramyl dipeptide). LTA-activated microglia kill co-cultured neurons apparently via nitric oxide, superoxide and peroxynitrite, which may induce apoptosis of neurons that are then phagocytosed by microglia." @default.
• W2072360147 created "2016-06-24" @default.
• W2072360147 creator A5038795824 @default.
• W2072360147 creator A5042066616 @default.
• W2072360147 date "2007-10-25" @default.
• W2072360147 modified "2023-09-26" @default.
• W2072360147 title "Neurodegeneration in models of Gram-positive bacterial infections of the central nervous system" @default.
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• W2072360147 doi "https://doi.org/10.1042/bst0351166" @default.
• W2072360147 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/17956303" @default.
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