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- W2072394974 endingPage "599" @default.
- W2072394974 startingPage "591" @default.
- W2072394974 abstract "As is the case for nearly every viral pathogen, non-enveloped viruses (NEV) must maintain their integrity under potentially harsh environmental conditions while retaining the ability to undergo rapid disassembly at the right time and right place inside host cells. NEVs generally exist in this metastable state until they encounter key cellular stimuli such as membrane receptors, decreased intracellular pH, digestion by cellular proteases, or a combination of these factors. These stimuli trigger conformational changes in the viral capsid that exposes a sequestered membrane-perturbing protein. This protein subsequently modifies the cell membrane in such a way as to allow passage of the virion and accompanying nucleic acid payload into the cell cytoplasm. Different NEVs employ variations of this general pathway for cell entry (Moyer and Nemerow, 2011, Curr. Opin. Virol., 1, 44-49), however this review will focus on significant new knowledge obtained on cell entry by human adenovirus (HAdV)." @default.
- W2072394974 created "2016-06-24" @default.
- W2072394974 creator A5027581149 @default.
- W2072394974 creator A5043937432 @default.
- W2072394974 date "2015-05-01" @default.
- W2072394974 modified "2023-10-17" @default.
- W2072394974 title "Adenovirus membrane penetration: Tickling the tail of a sleeping dragon" @default.
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