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• W2072411478 abstract "<i>Background:</i> Complement factor H (hCFH) plays a key inhibitory role in the control of the alternative complement pathway. We examined whether urinary hCFH (U-hCFH) levels is useful as an indirect indicator of renal damage. <i>Methods:</i> Urine samples were obtained from 104 patients with renal disease. Urine was collected with 10 m<i>M</i> EDTA and U-hCFH levels were measured using the BTA TRAK Assay Kit. <i>Results: </i>In the 62 patients with nephritis, the levels of U-hCFH were elevated (range 15–52,198 U/ml) over the normal range (0–14 U/ml). U-hCFH levels of patients with chronic renal failure, lupus nephritis, membranoproliferative glomerulonephritis, focal glomerulosclerosis were higher than that of IgA nephropathy patients (p < 0.05). In the patients with minimal change disease, showed high levels of U-hCFH during the nephrotic syndrome. U-hCFH was correlated significantly with urinary protein and urinary N-acetyl-β-<i>D</i>-glucosaminidase. <i>Conclusion:</i> We demonstrated that U-hCFH was detected in the urine of nephritis patients." @default.
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• W2072411478 date "2002-01-01" @default.
• W2072411478 modified "2023-10-15" @default.
• W2072411478 title "Urinary Complement Factor H in Renal Disease" @default.
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• W2072411478 doi "https://doi.org/10.1159/000064090" @default.
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