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• W2072604311 abstract "The mammalian UV response results in rapid and dramatic induction of c-jun. Induction of a protooncogene, normally involved in mitogenic responses, by a genotoxic agent that causes growth arrest seems paradoxical. We now provide an explanation for the role of c-Jun in the UV response of mouse fibroblasts. c-Jun is necessary for cell-cycle reentry of UV-irradiated cells, but does not participate in the response to ionizing radiation. Cells lacking c-Jun undergo prolonged cell-cycle arrest, but resist apoptosis, whereas cells that express c-Jun constitutively do not arrest and undergo apoptosis. This function of c-Jun is exerted through negative regulation of p53 association with the p21 promoter. Cells lacking c-Jun exhibit prolonged p21 induction, whereas constitutive c-Jun inhibits UV-mediated p21 induction." @default.
• W2072604311 created "2016-06-24" @default.
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• W2072604311 date "2000-12-01" @default.
• W2072604311 modified "2023-10-13" @default.
• W2072604311 title "The Mammalian UV Response" @default.
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• W2072604311 doi "https://doi.org/10.1016/s0092-8674(00)00193-8" @default.
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