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• W2072901766 abstract "Significance When neurons become active, they signal to local arterioles via intermediate glial cells, called astrocytes, to evoke dilation. This increases local blood flow and provides the oxygen and glucose necessary to support ongoing neuronal function. This process is termed neurovascular coupling. We demonstrate that chronic stress—which is a contributing factor for many diseases—impairs neurovascular coupling in the amygdala, a region involved in stressor processing. Our results further indicate that this dysfunction is due to the loss of arteriolar inwardly rectifying potassium (K + ) channel function, which makes vessels less able to respond to vasodilatory K + ions released by astrocytes during periods of increased neuronal activity. This neurovascular coupling impairment may contribute to the pathology of a range of brain disorders." @default.
• W2072901766 created "2016-06-24" @default.
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• W2072901766 date "2014-05-07" @default.
• W2072901766 modified "2023-10-10" @default.
• W2072901766 title "Stress-induced glucocorticoid signaling remodels neurovascular coupling through impairment of cerebrovascular inwardly rectifying K ⁺ channel function" @default.
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• W2072901766 doi "https://doi.org/10.1073/pnas.1401811111" @default.
• W2072901766 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/4034203" @default.
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• W2072901766 hasPublicationYear "2014" @default.
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