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- W2073082402 abstract "Regular physical activity is an important factor in the prevention and treatment of cardiovascular diseases. Post exercise hypotension (PEH) is a phenomenon in which a prolonged decrease in resting blood pressure occurs in the minutes and hours following acute exercise. However, the role of the phosphorylation of endothelial nitric oxide synthase (eNOS) on the PEH in hypertensive rats is not known. Therefore, this study is conducted to determine whether the phosphorylation of eNOS contributes to PEH in SHRs. To accomplish this, rats were subjected to a single bout of dynamic exercise on a treadmill at 15 m/min for 20 min. l-nitroarginine methyl ester (L-NAME, 40 mg/kg, i.p.) significantly inhibited post exercise hypotension (25±11 and 5±3mm Hg, respectively; P<0.05). In addition, the superoxide anion generation was decreased, while the plasma nitrite production and serine phosphorylation of endothelial nitric oxide synthase were significantly elevated in spontaneously hypertensive rats at 30 min after the termination of exercise. Taken together, these data demonstrate that the increased phosphorylation of endothelial nitric oxide synthase plays a crucial role in the reduction of arterial pressure following a single bout of dynamic exercise in spontaneously hypertensive rats." @default.
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- W2073082402 date "2009-10-01" @default.
- W2073082402 modified "2023-10-17" @default.
- W2073082402 title "Endothelial nitric oxide synthase activation is associated with post-exercise hypotension in spontaneously hypertensive rats" @default.
- W2073082402 doi "https://doi.org/10.1016/j.ijcard.2009.09.228" @default.
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